What are symptoms of uterine hyperstimulation that warranted stopping the medication?

Case Reports

. 1985 Sep;66(3 Suppl):16S-18S.

  • PMID: 2862618

Case Reports

Use of tocolytic drugs to reverse oxytocin-induced uterine hypertonus and fetal distress

J Lipshitz et al. Obstet Gynecol. 1985 Sep.

Abstract

The use of oxytocin in labor has the inherent danger of producing uterine hyperstimulation with resultant fetal distress. When produced by gradual titration of intravenous oxytocin, discontinuation of the medication is usually sufficient to reverse the process. However, the rapid administration of a large intravenous dose of oxytocin, as occurred in this patient, may result in hypertonic uterine contractions and fetal distress unresponsive to traditional measures. The rationale for using a tocolytic drug to reverse the uterine hypertonus, produce intrauterine fetal resuscitation, and prevent cesarean section is discussed in this report.

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A nurse is administering oxytocin to a client in labor. What are symptoms of uterine hyperstimulationthat would cause the nurse to discontinue this medication?

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What are symptoms of uterine hyperstimulation?

Symptoms.
Mild to moderate abdominal pain..
Abdominal bloating or increased waist size..
Nausea..
Vomiting..
Diarrhea..
Tenderness in the area of your ovaries..

What are symptoms of uterine hyperstimulation that warranted stopping oxytocin?

The main adverse effects of oxytocin are related to uterine hyperstimulation, where there's too much contraction. This could cause painful contractions, and lead to uterine rupture and hemorrhage. It could even restrict placental blood flow, resulting in abnormal fetal heart rate patterns.

How do you stop uterine hyperstimulation?

Uterine hyperstimulation may result in fetal heart rate abnormalities, uterine rupture, or placental abruption. It is usually treated by administering terbutaline.

What problems can result from uterine hyperstimulation?

Uterine hyperstimulation is a possibility when oxytocin is used to induce and augment labor. Unless the process is reversed, uteroplacental perfusion may decline; among the sequelae are fetal decelerations, bradycardia, placental abruption, and uterine rupture.