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Contents loading... Editors loading... Categories loading... When refering to evidence in academic writing, you should always try to reference the primary (original) source. That is usually the journal article where the information was first stated. In most cases Physiopedia articles are a secondary source and so should not be used as references. Physiopedia articles are best used to find the original sources of information (see the references list at the bottom of the article). If you believe that this Physiopedia article is the primary source for the information you are refering to, you can use the button below to access a related citation statement. Introduction[edit | edit source] Angiotensin Converting Enzyme (ACE) Inhibitors are a important class of drugs used in the management of cardiovascular and renal diseases, including Congestive Heart Failure (CHF), acute coronary syndrome, nephrotic syndrome, diabetes, and hypertension. Using an angiotensin-converting enzyme (ACE) inhibitor drug is an important part of treating heart failure. ACE inhibitors are strongly recommended as first-choice therapy in patients with heart failure[1]. Effects[edit | edit source]Angiotensin-converting enzyme inhibitors (ACEIs) improve heart failure by decreasing afterload, preload, and systolic wall stress, which results in increased cardiac output without any increase in heart rate. ACE inhibitors play an important role in:
Since the 1980s, several large, prospective, randomized, placebo-controlled trials have proved that treatment with ACE inhibitors reduces overall mortality, especially in patients with heart failure with reduced ejection fraction. Common Ace Inhibitors[edit | edit source]Several ACE inhibitors are on the market, and it is generally thought that they are equally beneficial in the treatment of heart failure. Commonly used ACE inhibitors include:
Implications for Physiotherapy[edit | edit source]Syncope AKA Fainting, swooning ACE inhibitors cause a reduction in blood pressure, this coupled with the naturally occurring post-exercise hypotension can result in excessive reductions in blood pressure. This can lead to dizziness and, in rare instances, syncope. Post-exercising clients on ACE inhibitors may need education in adhering to a gradual cool-down after each and every exercise session to prevent these symptoms and benefit the body by enhancing venous return and the prevention of blood pooling in the skeletal muscle. A gradual cool-down of five to 10 minutes of light aerobic activity allows the body to return to homeostasis and prevents excessive reductions in blood pressure.[3] References[edit | edit source]
What medications reduce preload?Preload reducers include NTG (eg, Deponit, Minitran, Nitro-Bid IV, Nitro-Bid ointment, Nitrodisc, Nitro-Dur, Nitrogard, Nitroglyn, Nitrol, Nitrolingual, Nitrong, Nitrostat, Transdermal-NTG, Transderm-Nitro, Tridil) and furosemide (eg, Lasix).
Which drug classification is used to decrease preload in patients with heart failure?Vasodilators decrease preload and/or afterload as well as reduce systemic vascular resistance (SVR).
Which medications reduce both preload and afterload in patients with heart failure?For heart failure, ACE inhibitors and ARBs reduce workload on the myocardium by reducing both preload and afterload.
What is a commonly prescribed drug for congestive heart failure?There are many different types of diuretic, but the most widely used for heart failure are furosemide (also called frusemide) and bumetanide.
Does hydralazine reduce preload?Hydralazine reduces afterload by directly dilating the arteries and long acting nitrates reduce preload by their vasodilator effects.
Which medications reduce afterload?Nifedipine and captopril are potent vasodilators and may be expected to help left ventricular failure by reducing afterload.
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Which medication would be prescribed to reduce preload for a patient with heart failure?
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